Abstracts 2.0

A majority of published scientific research is federally funded by taxpayer dollars in the U.S. yet most taxpayers have no idea why the research findings from these funds are important or how they contribute to a better society.

What if the article abstracts, laced with big words and jargon, were rewritten to a level where most people could understand; an abstract 2.o if you will? By reading a short summary of the work, anyone who wanted to know could actually understand the problem studied and the results. Maybe more importantly, the reader would not have to rely on interpretations of the research from popular media sources that have higher priorities than educating the public.

I have sat on this long enough. It’s not like a have anything else going on right now (except the birth of a son in a  month, syllabus to write, classes to prepare, evaluations to do, data to journal, …). Introducing:

Abstract 2.0

Here are the details presently. I and anyone willing to help will scour the journals of our respective fields and choose those we feel need to be disseminated to the larger public. In a short synopsis (abstract if you will), an overview of the article and why it is important will be written and deposited here. Details will be worked out on how to submit the abstracts in the near future.

Now is the time to act (or later if now is not convenient)!

Here is an example of a re-written abstract:

Colleen T. O’Loughlin, Laura C. Miller, Albert Siryaporn, Knut Drescher, Martin F. Semmelhack, and Bonnie L. Bassler (2013) 110:17981–17986, doi:10.1073/pnas.1316981110

 A quorum-sensing inhibitor blocks Pseudomonas aeruginosa virulence and biofilm formation

 Quorum sensing is a way a bacterium communicates to the cells around it to regulate behavior of the community as a whole. This process occurs in harmless bacteria as well as pathogens. One such pathogen, Pseudomonas aeruginosa, uses quorum sensing to attack its host in a concerted effort by all the cells present and to control how the cells ‘stick’ together once infecting the host. In an effort to prevent P. aeruginosa attack and infection, researchers tested synthetic molecules to identify those which block cells from receiving the attack message. One such molecule, meta-bromo-thiolactone (mBTL), succeeded in blocking the message and protected a roundworm model system and human lung cells from dying due to infection. The paper also discusses how mBTL works at the molecular level. The results from this study could help control complications in cystic fibrosis and hospital infections due to contaminated equipment.

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